TABLE 11

Summary of viabilities of hybrid progeny from D. melanogaster females and D. simulans males

GenotypeDosage of D. mel. Hmr+ aDosage of other Xme1 genes aViability bReferences
Xme1, Hmr/Xsim females01HighTables 3, 4 and 5
Xme1, In(1)AB/Xsim females<0.51Tables 3, 4 and 5
Xme1/Xsim ; +/Lhr females11Tables 1, 7
Xsim males00Sturtevant (1920); Tables 6, 8
Xme1, Hmr1/Xsim females≥0.51High, <29°Tables 2, 3, 5
Xme1; +/Lhr males22IntermediatecWatanabe (1979); Tables 1, 7
Xme1, In(1)AB males<12Hutter et al. (1990); Table 3
Xsim; Dp Hmr+ males20Tables 8, 10
Xme1/Xsim females11Sturtevant (1929), Kerkis (1933b), Watanabe et al. (1977), Lee (1978); Table 1
Xme1/Xsim; Dp Hmr+ females21LowcTable 7
Xme1, Hmr/Xme1 females12Table 6
Xme1, In(1)AB/Xme1 females<1.52Hutter et al. (1990); Table 6
Xme1, Hmr1 males≥12Hutter and Ashburner (1987); Tables 2, 3
Xme1, Hmr1/Xme1, Hmr+ females≥1.52LethalHutter et al. (1990); see also Dominant rescue of exceptional female hybrids in results.
Xme1/Xme1, females22Sturtevant (1920)
Xme1 males22Sturtevant (1920)
  • a Dosage calculations assume that X-linked genes are fully dosage compensated in hybrid males. See Hmr1 retains ≥50% of the activity of Hmr+ in discussion for the estimation of Hmr+ dosage for the Hmr1 and In(1)AB rescue alleles.

  • b The order of genotypes listed within each viability class is not significant.

  • c Viabilities of genotypes in these classes were often highly variable. See references for details.