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* School of Clinical Medical SciencesGerontology, University of Newcastle, Henry Wellcome Laboratory for Biogerontology Research, Newcastle upon Tyne, NE4 6BE, United Kingdom
Cancer Research United KingdomCentre for Cancer Therapeutics, Institute of Cancer Research, Sutton, Surrey, SM2 5NG, United Kingdom
1 Corresponding author: School of Clinical Medical SciencesGerontology, University of Newcastle, Henry Wellcome Laboratory for Biogerontology Research, Institute for Ageing and Health, Newcastle upon Tyne, NE4 6BE, United Kingdom.
E-mail: d.a.lydall{at}ncl.ac.uk
mutants. We conclude that Rad24 and Exo1 regulate nucleases with different properties at uncapped telomeres and propose a model to explain our findings. This article has been cited by other articles:
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M. P. Longhese DNA damage response at functional and dysfunctional telomeres Genes & Dev., January 15, 2008; 22(2): 125 - 140. [Abstract] [Full Text] [PDF] |
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J. F. Passos, G. Saretzki, and T. von Zglinicki DNA damage in telomeres and mitochondria during cellular senescence: is there a connection? Nucleic Acids Res., December 3, 2007; 35(22): 7505 - 7513. [Abstract] [Full Text] [PDF] |
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M. Hsu, M. J. McEachern, A. T. Dandjinou, Y. Tzfati, E. Orr, E. H. Blackburn, and N. F. Lue Telomerase core components protect Candida telomeres from aberrant overhang accumulation PNAS, July 10, 2007; 104(28): 11682 - 11687. [Abstract] [Full Text] [PDF] |
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