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Corresponding author: Maria Pia Longhese, Università di Milano-Bicocca, Piazza della Scienza 2, 20126 Milano, Italy., mariapia.longhese{at}unimib.it (E-mail)
Communicating editor: M. ROSE
mutants are defective in resuming DNA replication after transient deoxynucleotide depletion, and all display synthetic effects when also carrying mutations affecting the pol
-primase and RPA DNA replication complexes, suggesting a role for Bmh proteins in DNA replication stress response. Finally, the bmh1-169 bmh2
and bmh1-170 bmh2
mutants show increased rates of spontaneous gross chromosomal rearrangements, indicating that Bmh proteins are required to suppress genome instability.
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